New mechanisms for blood vessel growth identified as targets against drug-resistant tumors
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Researchers at CNIC and international collaborators have discovered alternative pathways that enable blood vessel growth without VEGF–VEGFR2 signaling. This finding reveals new potential targets for combating tumor angiogenesis, especially in drug-resistant cancers where current therapies targeting VEGF–VEGFR2 are ineffective.
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Originally published by gnews
